10.1.3

Biological & Social Explanations of GID

Test yourself

Biological Explanations of Gender Dysphoria

The causes of gender identity disorder have been researched. Findings suggest that it may be due to differing levels of sex hormones early in development. This theory has strengths and weaknesses.

Illustrative background for Brain functioningIllustrative background for Brain functioning ?? "content

Brain functioning

  • Brain functioning is slightly different between the sexes.
  • Those with gender identity disorder have brain functioning that is more typical of the sex that is not their biological sex.
  • In other words, the gender in which they identify.
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Hormones in the womb

  • Sex hormones like oestrogen and testosterone have impacts on the development of foetuses in the womb.
  • Generally speaking, higher levels of testosterone cause male genitalia to develop.
Illustrative background for Hormones and GIDIllustrative background for Hormones and GID ?? "content

Hormones and GID

  • If a person has unusual hormone levels during their development in the womb, they could develop brain function that is typical of that of their non-biological sex.
  • For example, a male could have high levels of oestrogen while developing.
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Kruijver et al (2000)

  • The bed nucleus of the stria terminalis (BSCt) was studied in male-to-female (MTF) transgenders by Kruijver et al (2000).
  • They found that the number of neurones in male to female individuals was similar to the number found in women.
  • The number is roughly half as many as men.
Illustrative background for __Hare et al (2009)__Illustrative background for __Hare et al (2009)__ ?? "content

Hare et al (2009)

  • Androgens are male sex hormones; the major example is testosterone.
  • Hare et al (2009) studied male-to-female transgender (male to female) and found they were more likely to have a particular gene that coded for an androgen receptor.
  • They compare male to female individuals to non-transgender males.
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Support

  • Both these studies support biological explanations of gender identity disorder.
  • It is important to note that in both of these studies, the transsexual people were mostly living as their identified gender (non-biological sex) and some of them had changed their physical sex.

Weaknesses with the Biological Theory of Gender Identity Disorder

It has been suggested that GID may be due to differing levels of sex hormones early in development. But some people have found weaknesses with this proposition.

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Cause and effect

  • Another set of researchers studied the bed nucleus of the stria terminalis (BSCt).
  • Chung et al. (2002) stated that the differences in sexes of the BSCt did not appear until adulthood.
  • So the BSCt differences could be the result of gender development, not the cause.
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Validity

  • Congenital adrenal hyperplasia (CAH) is a syndrome whereby males or females produce significantly more testosterone than the norm.
  • You might expect girls with CAH to have gender identity disorder because they have higher amounts of testosterone.
  • But most girls with CAH do not develop GID.
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Limitations

  • The theory does not take into account other factors that may have an impact on gender development, like social learning.

Social Explanation of Gender Identity Disorder

One social explanation of GID is the social learning theory (SLT), whereby children imitate role models on how to act in terms of gender. Several studies have been done, with some controversial conclusions.

Illustrative background for Social learning theoryIllustrative background for Social learning theory ?? "content

Social learning theory

  • Social learning theory proposes that learning happens through observing and imitating the behaviour of a role model.
Illustrative background for SLT and GIDIllustrative background for SLT and GID ?? "content

SLT and GID

  • This theory can be used to explain how gender identity disorder develops.
  • If a male child, for example, imitates behaviours of a female role model, they may develop a female gender that does not match with their biological sex.
  • The behaviour has to be reinforced (for example positively through praise or rewards) for the behaviours to continue.
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Rekers and Lovaas (1974)

  • A controversial study was published in 1974 by Rekers and Lovaas which claimed that gender identity disorder can be ‘cured’ by reinforcing the gender behaviours of the corresponding biological sex.
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The case study

  • The evidence of this was a case study of a boy who displayed feminine behaviours.
  • The mother (trained to be his therapist) reinforced masculine traits in the boy while attempting to minimise feminine traits.
  • This was done both in a clinic and at home.
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Results

  • The results showed that after three years, the boy still displayed masculine traits.
  • So, the boy was ‘normalised’ and the treatment deemed a success.
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Controversy

  • This study was controversial as many psychologists do not agree that gender identity disorder should be cured.
  • This idea causes stigma to those with atypical gender identity.
Illustrative background for __Rekers (1977)__Illustrative background for __Rekers (1977)__ ?? "content

Rekers (1977)

  • Rekers (1977) suggested that gender identity disorder should be ‘cured’ and that genders should match biological sex. - He argued that the same should be done with homosexuality, which was labelled a disorder that could be ‘cured’.

Jump to other topics

1Social Influence

1.1Social Influence

1.1.1Conformity

1.1.2Asch (1951)

1.1.3Sherif (1935)

1.1.4Conformity to Social Roles

1.1.5BBC Prison Study

1.1.6End of Topic Test - Conformity

1.1.7Obedience

1.1.8Analysing Milgram's Experiment

1.1.9Agentic State & Legitimate Authority

1.1.10Variables of Obedience

1.1.11Resistance to Social Influence

1.1.12Minority Influence & Social Change

1.1.13Minority Influence & Social Impact Theory

1.1.14End of Topic Test - Social Influences

1.1.15Exam-Style Question - Conformity

1.1.16Top Grade AO2/AO3 - Social Influence

2Memory

2.1Memory

2.1.1Multi-Store Model of Memory

2.1.2Short-Term vs Long-Term Memory

2.1.3Long-Term Memory

2.1.4Support for the Multi-Store Model of Memory

2.1.5Duration Studies

2.1.6Capacity Studies

2.1.7Coding Studies

2.1.8The Working Memory Model

2.1.9The Working Memory Model 2

2.1.10Support for the Working Memory Model

2.1.11Explanations for Forgetting

2.1.12Studies on Interference

2.1.13Cue-Dependent Forgetting

2.1.14Eye Witness Testimony - Loftus & Palmer

2.1.15Eye Witness Testimony Loftus

2.1.16Eyewitness Testimony - Post-Event Discussion

2.1.17Eyewitness Testimony - Age & Misleading Questions

2.1.18Cognitive Interview

2.1.19Cognitive Interview - Geiselman & Fisher

2.1.20End of Topic Test - Memory

2.1.21Exam-Style Question - Memory

2.1.22A-A* (AO3/4) - Memory

3Attachment

3.1Attachment

3.1.1Caregiver-Infant Interaction

3.1.2Condon & Sander (1974)

3.1.3Schaffer & Emerson (1964)

3.1.4Multiple Attachments

3.1.5Studies on the Role of the Father

3.1.6Animal Studies of Attachment

3.1.7Explanations of Attachment

3.1.8Attachment Types - Strange Situation

3.1.9Cultural Differences in Attachment

3.1.10Disruption of Attachment

3.1.11Disruption of Attachment - Privation

3.1.12Overcoming the Effects of Disruption

3.1.13The Effects of Institutionalisation

3.1.14Early Attachment

3.1.15Critical Period of Attachment

3.1.16End of Topic Test - Attachment

3.1.17Exam-Style Question - Attachment

3.1.18Top Grade AO2/AO3 - Attachment

4Psychopathology

4.1Psychopathology

4.1.1Definitions of Abnormality

4.1.2Definitions of Abnormality 2

4.1.3Phobias, Depression & OCD

4.1.4Phobias: Behavioural Approach

4.1.5Evaluation of Behavioural Explanations of Phobias

4.1.6Depression: Cognitive Approach

4.1.7OCD: Biological Approach

4.1.8Evidence for the Biological Approach

4.1.9End of Topic Test - Psychopathy

4.1.10Exam-Style Question - Phobias

4.1.11Top Grade AO2/AO3 - Psychopathology

5Approaches in Psychology

5.1Approaches in Psychology

5.1.1Psychology as a Science

5.1.2Origins of Psychology

5.1.3Reductionism & Problems with Introspection

5.1.4The Behaviourist Approach - Classical Conditioning

5.1.5Pavlov's Experiment

5.1.6Little Albert Study

5.1.7The Behaviourist Approach - Operant Conditioning

5.1.8Social Learning Theory

5.1.9The Cognitive Approach 1

5.1.10The Cognitive Approach 2

5.1.11The Biological Approach

5.1.12Gottesman (1991) - Twin Studies

5.1.13Brain Scanning

5.1.14Structure of Personality & Little Hans

5.1.15The Psychodynamic Approach (A2 only)

5.1.16Humanistic Psychology (A2 only)

5.1.17Aronoff (1957) (A2 Only)

5.1.18Rogers' Client-Centred Therapy (A2 only)

5.1.19End of Topic Test - Approaches in Psychology

5.1.20Exam-Style Question - Approaches in Psychology

5.2Comparison of Approaches (A2 only)

5.2.1Psychodynamic Approach

5.2.2Cognitive Approach

5.2.3Biological Approach

5.2.4Behavioural Approach

5.2.5End of Topic Test - Comparison of Approaches

6Biopsychology

6.1Biopsychology

6.1.1Nervous System Divisions

6.1.2Neuron Structure & Function

6.1.3Neurotransmitters

6.1.4Endocrine System Function

6.1.5Fight or Flight Response

6.1.6The Brain (A2 only)

6.1.7Localisation of Brain Function (A2 only)

6.1.8Studying the Brain (A2 only)

6.1.9CIMT (A2 Only) & Postmortem Examinations

6.1.10Biological Rhythms (A2 only)

6.1.11Studies on Biological Rhythms (A2 Only)

6.1.12End of Topic Test - Biopsychology

6.1.13Top Grade AO2/AO3 - Biopsychology

7Research Methods

7.1Research Methods

7.1.1Experimental Method

7.1.2Observational Techniques

7.1.3Covert, Overt & Controlled Observation

7.1.4Self-Report Techniques

7.1.5Correlations

7.1.6Exam-Style Question - Research Methods

7.1.7End of Topic Test - Research Methods

7.2Scientific Processes

7.2.1Aims, Hypotheses & Sampling

7.2.2Pilot Studies & Design

7.2.3Questionnaires

7.2.4Variables & Control

7.2.5Demand Characteristics & Investigator Effects

7.2.6Ethics

7.2.7Limitations of Ethical Guidelines

7.2.8Consent & Protection from Harm Studies

7.2.9Peer Review & The Economy

7.2.10Validity (A2 only)

7.2.11Reliability (A2 only)

7.2.12Features of Science (A2 only)

7.2.13Paradigms & Falsifiability (A2 only)

7.2.14Scientific Report (A2 only)

7.2.15Scientific Report 2 (A2 only)

7.2.16End of Topic Test - Scientific Processes

7.3Data Handling & Analysis

7.3.1Types of Data

7.3.2Descriptive Statistics

7.3.3Correlation

7.3.4Evaluation of Descriptive Statistics

7.3.5Presentation & Display of Data

7.3.6Levels of Measurement (A2 only)

7.3.7Content Analysis (A2 only)

7.3.8Case Studies (A2 only)

7.3.9Thematic Analysis (A2 only)

7.3.10End of Topic Test - Data Handling & Analysis

7.4Inferential Testing

7.4.1Introduction to Inferential Testing

7.4.2Sign Test

7.4.3Piaget Conservation Experiment

7.4.4Non-Parametric Tests

8Issues & Debates in Psychology (A2 only)

8.1Issues & Debates in Psychology (A2 only)

8.1.1Culture Bias

8.1.2Sub-Culture Bias

8.1.3Gender Bias

8.1.4Ethnocentrism

8.1.5Cross Cultural Research

8.1.6Free Will & Determinism

8.1.7Comparison of Free Will & Determinism

8.1.8Reductionism & Holism

8.1.9Reductionist & Holistic Approaches

8.1.10Nature-Nurture Debate

8.1.11Interactionist Approach

8.1.12Nature-Nurture Methods

8.1.13Nature-Nurture Approaches

8.1.14Idiographic & Nomothetic Approaches

8.1.15Socially Sensitive Research

8.1.16End of Topic Test - Issues and Debates

9Option 1: Relationships (A2 only)

9.1Relationships: Sexual Relationships (A2 only)

9.1.1Sexual Selection & Human Reproductive Behaviour

9.1.2Intersexual & Intrasexual Selection

9.1.3Evaluation of Sexual Selection Behaviour

9.1.4Factors Affecting Attraction: Self-Disclosure

9.1.5Evaluation of Self-Disclosure Theory

9.1.6Self Disclosure in Computer Communication

9.1.7Factors Affecting Attraction: Physical Attributes

9.1.8Matching Hypothesis Studies

9.1.9Factors Affecting Physical Attraction

9.1.10Factors Affecting Attraction: Filter Theory 1

9.1.11Factors Affecting Attraction: Filter Theory 2

9.1.12Evaluation of Filter Theory

9.1.13End of Topic Test - Sexual Relationships

9.2Relationships: Romantic Relationships (A2 only)

9.2.1Social Exchange Theory

9.2.2Evaluation of Social Exchange Theory

9.2.3Equity Theory

9.2.4Evaluation of Equity Theory

9.2.5Rusbult’s Investment Model

9.2.6Evaluation of Rusbult's Investment Model

9.2.7Relationship Breakdown

9.2.8Studies on Relationship Breakdown

9.2.9Evaluation of Relationship Breakdown

9.2.10End of Topic Test - Romantic relationships

9.3Relationships: Virtual & Parasocial (A2 only)

9.3.1Virtual Relationships in Social Media

9.3.2Evaluation of Reduced Cues & Hyperpersonal

9.3.3Parasocial Relationships

9.3.4Attachment Theory & Parasocial Relationships

9.3.5Evaluation of Parasocial Relationship Theories

9.3.6End of Topic Test - Virtual & Parasocial Realtions

10Option 1: Gender (A2 only)

10.1Gender (A2 only)

10.1.1Sex, Gender & Androgyny

10.1.2Gender Identity Disorder

10.1.3Biological & Social Explanations of GID

10.1.4Biological Influences on Gender

10.1.5Effects of Hormones on Gender

10.1.6End of Topic Test - Gender 1

10.1.7Kohlberg’s Theory of Gender Constancy

10.1.8Evaluation of Kohlberg's Theory

10.1.9Gender Schema Theory

10.1.10Psychodynamic Approach to Gender Development 1

10.1.11Psychodynamic Approach to Gender Development 2

10.1.12Social Approach to Gender Development

10.1.13Criticisms of Social Theory

10.1.14End of Topic Test - Gender 2

10.1.15Media Influence on Gender Development

10.1.16Cross Cultural Research

10.1.17Childcare & Gender Roles

10.1.18End of Topic Test - Gender 3

11Option 1: Cognition & Development (A2 only)

11.1Cognition & Development (A2 only)

11.1.1Piaget’s Theory of Cognitive Development 1

11.1.2Piaget's Theory of Cognitive Development 2

11.1.3Schema Accommodation Assimilation & Equilibration

11.1.4Piaget & Inhelder’s Three Mountains Task (1956)

11.1.5Conservation & Class Inclusion

11.1.6Evaluation of Piaget

11.1.7End of Topic Test - Cognition & Development 1

11.1.8Vygotsky

11.1.9Evaluation of Vygotsky

11.1.10Baillargeon

11.1.11Baillargeon's studies

11.1.12Evaluation of Baillargeon

11.1.13End of Topic Test - Cognition & Development 2

11.1.14Sense of Self & Theory of Mind

11.1.15Baron-Cohen Studies

11.1.16Selman’s Five Levels of Perspective Taking

11.1.17Biological Basis of Social Cognition

11.1.18Evaluation of Biological Basis of Social Cognition

11.1.19Important Issues in Social Neuroscience

11.1.20End of Topic Test - Cognition & Development 3

11.1.21Top Grade AO2/AO3 - Cognition & Development

12Option 2: Schizophrenia (A2 only)

12.1Schizophrenia: Diagnosis (A2 only)

12.1.1Classification & Diagnosis

12.1.2Reliability & Validity of Diagnosis

12.1.3Gender & Cultural Bias

12.1.4Pinto (2017) & Copeland (1971)

12.1.5End of Topic Test - Scizophrenia Diagnosis

12.2Schizophrenia: Treatment (A2 only)

12.2.1Family-Based Psychological Explanations

12.2.2Evaluation of Family-Based Explanations

12.2.3Cognitive Explanations

12.2.4Drug Therapies

12.2.5Evaluation of Drug Therapies

12.2.6Biological Explanations for Schizophrenia

12.2.7Dopamine Hypothesis

12.2.8End of Topic Test - Schizoprenia Treatment 1

12.2.9Psychological Therapies 1

12.2.10Psychological Therapies 2

12.2.11Evaluation of Psychological Therapies

12.2.12Interactionist Approach - Diathesis-Stress Model

12.2.13Interactionist Approach - Triggers & Treatment

12.2.14Evaluation of the Interactionist Approach

12.2.15End of Topic Test - Scizophrenia Treatments 2

13Option 2: Eating Behaviour (A2 only)

13.1Eating Behaviour (A2 only)

13.1.1Explanations for Food Preferences

13.1.2Birch et al (1987) & Lowe et al (2004)

13.1.3Control of Eating Behaviours

13.1.4Control of Eating Behaviour: Leptin

13.1.5Biological Explanations for Anorexia Nervosa

13.1.6Psychological Explanations: Family Systems Theory

13.1.7Psychological Explanations: Social Learning Theory

13.1.8Psychological Explanations: Cognitive Theory

13.1.9Biological Explanations for Obesity

13.1.10Biological Explanations: Studies

13.1.11Psychological Explanations for Obesity

13.1.12Psychological Explanations: Studies

13.1.13End of Topic Test - Eating Behaviour

14Option 2: Stress (A2 only)

14.1Stress (A2 only)

14.1.1Physiology of Stress

14.1.2Role of Stress in Illness

14.1.3Role of Stress in Illness: Studies

14.1.4Social Readjustment Rating Scales

14.1.5Hassles & Uplifts Scales

14.1.6Stress, Workload & Control

14.1.7Stress Level Studies

14.1.8End of Topic Test - Stress 1

14.1.9Physiological Measures of Stress

14.1.10Individual Differences

14.1.11Stress & Gender

14.1.12Drug Therapy & Biofeedback for Stress

14.1.13Stress Inoculation Therapy

14.1.14Social Support & Stress

14.1.15End of Topic Test - Stress 2

15Option 3: Aggression (A2 only)

15.1Aggression: Physiological (A2 only)

15.1.1Neural Mechanisms

15.1.2Serotonin

15.1.3Hormonal Mechanisms

15.1.4Genetic Factors

15.1.5Genetic Factors 2

15.1.6End of Topic Test - Aggression: Physiological 1

15.1.7Ethological Explanation

15.1.8Innate Releasing Mechanisms & Fixed Action Pattern

15.1.9Evolutionary Explanations

15.1.10Buss et al (1992) - Sex Differences in Jealousy

15.1.11Evaluation of Evolutionary Explanations

15.1.12End of Topic Test - Aggression: Physiological 2

15.2Aggression: Social Psychological (A2 only)

15.2.1Social Psychological Explanation

15.2.2Buss (1963) - Frustration/Aggression

15.2.3Social Psychological Explanation 2

15.2.4Social Learning Theory (SLT) 1

15.2.5Social Learning Theory (SLT) 2

15.2.6Limitations of Social Learning Theory (SLT)

15.2.7Deindividuation

15.2.8Deindividuation 2

15.2.9Deindividuation - Diener et al (1976)

15.2.10End of Topic Test - Aggression: Social Psychology

15.2.11Institutional Aggression: Prisons

15.2.12Evaluation of Dispositional & Situational

15.2.13Influence of Computer Games

15.2.14Influence of Television

15.2.15Evaluation of Studies on Media

15.2.16Desensitisation & Disinhibition

15.2.17Cognitive Priming

15.2.18End of Topic Test - Aggression: Social Psychology

16Option 3: Forensic Psychology (A2 only)

16.1Forensic Psychology (A2 only)

16.1.1Defining Crime

16.1.2Measuring Crime

16.1.3Offender Profiling

16.1.4Evaluation of Offender Profiling

16.1.5John Duffy Case Study

16.1.6Biological Explanations 1

16.1.7Biological Explanations 2

16.1.8Evaluation of the Biological Explanation

16.1.9Cognitive Explanations

16.1.10Moral Reasoning

16.1.11Psychodynamic Explanation 1

16.1.12Psychodynamic Explanation 2

16.1.13End of Topic Test - Forensic Psychology 1

16.1.14Differential Association Theory

16.1.15Custodial Sentencing

16.1.16Effects of Prison

16.1.17Evaluation of the Effects of Prison

16.1.18Recidivism

16.1.19Behavioural Treatments & Therapies

16.1.20Effectiveness of Behavioural Treatments

16.1.21Restorative Justice

16.1.22End of Topic Test - Forensic Psychology 2

17Option 3: Addiction (A2 only)

17.1Addiction (A2 only)

17.1.1Definition

17.1.2Brain Neurochemistry Explanation

17.1.3Learning Theory Explanation

17.1.4Evaluation of a Learning Theory Explanation

17.1.5Cognitive Bias

17.1.6Griffiths on Cognitive Bias

17.1.7Evaluation of Cognitive Theory (A2 only)

17.1.8End of Topic Test - Addiction 1

17.1.9Gambling Addiction & Learning Theory

17.1.10Social Influences on Addiction 1

17.1.11Social Influences on Addiction 2

17.1.12Personal Influences on Addiction

17.1.13Genetic Explanations of Addiction

17.1.14End of Topic Test - Addiction 2

17.2Treating Addiction (A2 only)

17.2.1Drug Therapy

17.2.2Behavioural Interventions

17.2.3Cognitive Behavioural Therapy

17.2.4Theory of Reasoned Action

17.2.5Theory of Planned Behaviour

17.2.6Six Stage Model of Behaviour Change

17.2.7End of Topic Test - Treating Addiction

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Gender Identity Disorder

Biological Influences on Gender